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It has been shown to induce hippocampal oxidative stress, a neuroinflammatory response, neuronal death via apoptosis, and ultimately memory impairment, and has been widely used in preclinical models to investigate postoperative cognitive dysfunction [4, 6, 30]

Its strong safety profile and effective absorption make it suitable for a wide range of patients, including those with poor dietary intake, malabsorption issues, or conditions affecting intrinsic factor

Open-label 24-week extension study of edaravone (MCI-186) in amyotrophic lateral sclerosis

FIGURE 9 The core of this toxicity mechanism lies in the depletion of the labile Fe 2+ pool (Figure 5B)without altering total cellular iron (Figure 5A)which triggers a compensatory iron-starvation response (IREB2, TfR1, Ferritin